https://nova.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:8429 Sat 24 Mar 2018 08:40:48 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:8132 Sat 24 Mar 2018 08:40:03 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:16020 0.46, p<0.05). Conclusions: Older schizophrenia patients show a peculiar pattern of CSF Abeta42 and tau concentrations that relates to cognitive and structural markers but is not consistent with neurodegeneration and could be secondary to neurodevelopmental or drug treatment effects.]]> Sat 24 Mar 2018 08:19:31 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:19040 Sat 24 Mar 2018 08:05:18 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:17935 0.29). Hippocampal surface analysis showed the typical pattern of CA1 and subicular tissue atrophy in both ε4-carriers and non-carriers compared with controls (e4 carriers: p < 0.0002; ε4 non-carriers: p < 0.01, permutation test). The left hippocampal volume was significantly smaller in ε4-carriers than non-carriers (p = 0.044, Mann–Whitney test), the effect of APOE4 mapping to the subicular/CA1 region (p = 0.041, permutation test). Differences were not statistically significant in the right hippocampus (p > 0.20, permutation test). These findings show that hippocampal atrophy is greater in APOE4 carriers in regions typically affected by pathology. APOE4 may affect the structural expression of Alzheimer's disease.]]> Sat 24 Mar 2018 07:56:34 AEDT ]]>